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These adaptations range from altered neurotransmitter levels to altered cell morphology and changes in transcriptional activity [ 15 ]. Drugs and natural reward One issue that needs verification is whether drugs and natural rewards activate the same populations of neurons. For example, sugar and saccharin can reduce self-administration of cocaine and heroin [ 7 ]. This was inspired from studies on the role of cellular events underlying Synaptic Plasticity processes of learning and the behavioral effects of drugs [ 10 ]. Due to this requirement, it has been proposed that there exists a single neural system which processes rewards of all modalities and thereby functions as a common scale through which diverse rewards may be contrasted [ 3 ]. This article has been cited by other articles in PMC. This theory can be recognized as an extension of the incentive-sensitisation theory which hypothesizes how drugs interact with natural rewards. Based on previous data, can we understand drug addiction through studying natural reward? Money, which allows us access to food and improves our chance for reproduction, is a more abstract reward. However, here we discuss current evidence for the orquestic regulation of the different neurotransmitters on reward-seeking behavior and its potential effect on drug addiction. In addition to relying on strong neuropsychopharmacological data, the ADM provides an original conception of motivational specificity. By Synaptic plasticity we mean alterations at the level of the synapse, typically measured using electrophysiological methods e.
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